Study reveals previously unknown mechanisms in the kidney that control magnesium and calcium levels
A mutation in the gene KCTD1 leads to kidney defects that can ultimately cause metabolic bone disease.
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Dr. Marneros is an Associate Professor of Dermatology at Harvard Medical School and a Principal Investigator at the Cutaneous Biology Research Center of Massachusetts General Hospital. He is a board-certified dermatologist with a clinical interest in general and medical dermatology, skin cancer and genetic diseases of the skin. Born in Germany, he obtained his MD from the University of Cologne and did a medical doctoral thesis on dermatologic genetics at Harvard Medical School. He obtained a PhD in Cell Biology under the supervision of Dr. Bjorn Olsen at Harvard Medical School, followed by a postdoctoral fellowship in Vascular Biology again at Harvard Medical School. After his postdoctoral training he completed an academic-track Dermatology residency at New York Presbyterian Medical Center of Columbia University in New York City. Dr. Marneros is now leading an NIH-funded research laboratory at the Cutaneous Biology Research Center (CBRC) and is clinically active in the Dermatology Clinics of Massachusetts General Hospital.
He is an expert in vascular biology, skin cancer and genetic diseases of the skin, which is documented in numerous peer-reviewed publications and textbook chapters. As a physician-scientist his translational research is aimed at better understanding dermatologic diseases and improving patient care. To learn more about his research visit the Marneros Lab at the CBRC Center.
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A mutation in the gene KCTD1 leads to kidney defects that can ultimately cause metabolic bone disease.
New research by investigators at Massachusetts General Hospital (MGH) reveals insights into potential drivers of the disease — which currently has no cure — that could be targeted through prevention or treatment strategies. The findings are published in eLife.