About Alexander Faje, MD

Alexander Faje, MD is a clinician in the Neuroendocrine Clinical Center and a faculty member in the Neuroendocrine Unit at the Massachusetts General Hospital, and an Instructor of Medicine at Harvard Medical School.  He is a clinical researcher and clinician, with a focus on pituitary disease.

Departments, Centers, & Programs:



Mass General Endocrinology: Neuroendocrine & Pituitary Tumor Clinical Center
55 Fruit St.
Boston, MA 02114
Phone: 617-726-7948

Medical Education

  • MD, Vanderbilt University School of Medicine
  • Residency, University of Michigan Medical Center
  • Fellowship, Massachusetts General Hospital

American Board Certifications

  • Endocrinology, Diabetes and Metabolism, American Board of Internal Medicine

Accepted Insurance Plans

Note: This provider may accept more insurance plans than shown; please call the practice to find out if your plan is accepted.



    High-dose glucocorticoids for the treatment of ipilimumab-induced hypophysitis is associated with reduced survival in patients with melanoma. Cancer 2018

    Dopamine agonists can reduce cystic prolactinomas. J Clin Endocrinol Metab 2016

    Immunotherapy and hypophysitis: clinical presentation, treatment, and biologic insights. Pituitary 2016

    Neuroendocrine disorders: pituitary imaging. Handb Clin Neurol 2016

    Hypophysitis: evaluation and management. Clin Diabetes Endocrinol 2016

    Ipilimumab-induced hypophysitis: a detailed longitudinal analysis in a large cohort of patients with metastatic melanoma. J Clin Endocrinol Metab 2014

    The treatment of hyperprolactinemia in postmenopausal women with prolactin-secreting microadenomas: cons. Endocrine 2014

    Central diabetes insipidus: a previously unreported side effect of temozolomide. J Clin Endocrinol Metab 2013

    Current treatment options for hyperprolactinemia. Expert Opin Pharmacother 2013

    Cavernous carotid artery aneurysm, a rare cause of intrasellar mass and hyperprolactinemia. J Clin Endocrinol Metab 2012

    Basal, but not pulsatile, GH secretion determines the ambient circulating levels of IGF-1. J Clin Endocrinol Metab 2010

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